WHAT DOES SPINOCEREBELLAR ATAXIA TREATMENT MEAN?

In 1993, collaborative attempts from the laboratories of Drs. Harry T. Orr and Huda Y. Zoghbi found that SCA1 is due to the growth of a CAG repeat somewhere inside a client’s DNA. CAG repeats trigger a polyglutamine growth in the protein that the mutated gene encodes; in this case, the team later recognized this experienced transpired in Ataxin-1

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